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I just got some new brain research on my hands.

The latest research, conducted by a team at Emory University, shows that the brain’s ability to store information has been impaired in some patients with epilepsy.

The study, published in The Journal of Neurosurgery, found that patients with the condition have less than half the amount of neurons in their brains as normal.

The researchers theorize that the loss of the neurons could impair the brain in a number of ways.

One way would be to slow down the brain.

A second would be through the disruption of the connections between neurons.

That could lead to abnormal firing patterns in the neurons.

Finally, there’s a third possible effect, which the researchers speculate may be the most important.

The brain’s immune system could be working against the brain when it’s being exposed to toxins or infections.

The study is the first to examine how this disorder affects a person’s ability and ability to learn.

“In the last few years, we’ve seen that there are a number neurological diseases that affect learning,” said lead author Dr. Daniela L. DiMaggio, who is a neurosurgeon at Emry University.

“But until now, we didn’t know if this was the case in the brain, how it affected learning, and what it meant for patients.”

DiMaggios study is a pilot study to determine whether the disorder impacts learning in patients with chronic seizures, and it was conducted with a large number of people.

They recruited 14 patients with one of the conditions.

All of the patients underwent two rounds of MRI scans and were then given a dose of a medication called ketamine, which acts on a different receptor than the one that the seizure medications affect.

The patients in the study were not prescribed ketamine and did not receive the medication in the baseline scans.

After the ketamine treatment, the researchers used a brain imaging technique called functional magnetic resonance imaging (fMRI) to look at the brain activity of each patient.

The imaging showed that the patients’ brains showed activity that correlated with their seizure-induced brain activity, and the brain was still firing after the ketamines had been administered.

This pattern of activity is called “synaptic plasticity,” and it’s a hallmark of the brain that allows neurons to send and receive information.

DiMags study is just the latest research to indicate that the neurological disease known as epilepsy affects learning in the patients.

In 2012, researchers found that epilepsy patients had a decreased ability to process information and retain it as new information.

Researchers theorized that this lack of processing could impair neural connections, and therefore the ability to remember new information, even after patients had received the medication for their seizures.

But there’s more to the story than just the lack of new information and synaptic plasticity.

Di Maggios team found that when patients with this disorder received ketamine for their epilepsy, their brains had a higher level of electrical activity than their brains without epilepsy.

And when they received ketamines for the epilepsy and were given the medication, their electrical activity also increased, indicating that they had more synapses than healthy brains.

This study adds to a growing body of evidence that the neurons that process the signals sent by the brain are damaged in some people with epilepsy, but researchers have been unable to pinpoint exactly what is causing the damage.

The new study adds further evidence that seizures are a risk factor for learning and memory loss.

“We think it’s very likely that the ketone supplementation of epilepsy patients is a potential tool to help them get better, and that the results from this study suggest that this is a promising approach to help patients with seizures,” said Dr. Michael E. Schulze, a neurologist at Emrys University.

In the future, the team hopes to test whether ketamine will work in other patients who are also on medication for epilepsy, and if it could be used to treat other neurological conditions.

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